Original Research Article
ABSTRACT
Background: Gas stations are areas that operate in the petroleum sector where repeated exposure to the products produced (butane, pentane, isopentane, benzene, alkylbenzene, toluene, and xylene) can have an impact in the form of health problems. The individuals with the greatest exposure to gas station products are the workers. According to data, during the period from 2016 to 2018 there were 120 cases of work accidents around gas stations. The use of Personal Protective Equipment (PPE) and training regarding the prevention for the worker and management of accidents and illnesses resulting from working at gas stations are important in ensuring worker safety. According to research that has been conducted, it was found that the use of Personal Protective Equipment (PPE) is still very low among Gas Station X workers, Maulafa District, Kupang City. Training and equipment that support Occupational Safety and Health programs are also not available to the majority of workers. This is the cause of health problems among Gas Station X workers, Maulafa District, Kupang City. Method: This research is a quantitative study with a cross sectional approach and involved 12 respondents. This study data collection techniques by interview dan physical examination directly to the samples at Gas Station X Maulafa District, Kupang City. Result: Based on the research results, it was found that the majority of Gas Station The health problems found were Peripheral Vertigo (16%), Tension Type Headache (TTH) (8%), Allergic Rhinitis (8%) and Irritant Contact Dermatitis (8%). Conclusion: An occupational risk factor that can affect the occupational safety and health of employees at GAS STATION X is exposure to fuel oil which contains volatile substances. It was found that 4 out of 12 workers at the gas station in Maulafa District, Kupang City experienced health problems, the health problems they experienced were Peripheral Vertigo, Tension Type Headache, Allergic Rhinitis and Irritant ...........
Case Report
Rexpansion Pulmonary Edema
A. Tissir, Y. Haouas, Z. Allal, S. Chabbar, Fz. Faouji, A. Mounir, C. Elkettani, L. Barrou
EAS J Anesthesiol Crit Care; 2024, 6(3): 36-38
DOI: 10.36349/easjacc.2024.v06i03.002
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ABSTRACT
Ex vacuo pulmonary edema is a rare but potentially severe event with a mortality of 15-20%. We report the case of a patient who presented with ex vacuo pulmonary edema after drainage of a right pneumothorax. The pathophysiology of ex vacuo pulmonary edema is not yet clearly established, with mechanical and inflammatory factors (production of interleukin 8 and leukotriene B4) being the most incriminating. Certain factors, such as the duration and extent of pulmonary collapse and the speed of lung re-expansion after drainage, appear to favour the onset of ex vacuo edema, justifying certain precautionary measures. Curative treatment relies essentially on oxygenation and reduction of pleural aspiration pressures, but the best treatment is prevention.
Original Research Article
ABSTRACT
The death is a major accident of which the assessment and analysis are needed to improve the quality of care in an ICU. The aim of our work was to study mortality, analyze the main causes and assess avoidable and non-avoidable causes, in order to target possible preventive action. Prospective study carried out in the surgical intensive care unit, including all patients who died more than 48 hours after admission. Several severity scores were tested (Apache, SOFA, IGS), but none of them showed superiority over the others, and therefore represent only estimates of the severity of multivisceral failure. This high mortality rate can be explained by several factors: age, causal pathology and associated defects, but also by nosocomial infections, which remain a major cause and which can be reversed by preventive measures, in particular the correct prescription of antibiotics.
ABSTRACT
Myasthenia gravis (MG) is an autoimmune disorder affecting the neuromuscular junction caused by a B-cell-mediated, T-cell-dependent immunologic attack at the end plate of the postsynaptic membrane. Attack on muscle acetylcholine receptors (AChR) of the postsynaptic membrane due to the AChR, muscle-specific tyrosine kinase, or lipoprotein receptor-related peptide 4 antibodies lead to symptoms of painless, fluctuating weakness of muscle groups and often begins with ocular signs and symptoms. Coronavirus disease 2019 (COVID-19) is an acute respiratory disease caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), a novel coronavirus closely related to SARS-CoV. Serious neurologic complications are infrequent and diverse with reported cases of stroke, encephalitis/meningitis, Guillain-Barré syndrome, acute disseminated encephalomyelitis, ataxia, and unspecified limb weakness. MG is a rarely reported sequela of COVID-19 infection. To date, there are 15 reported cases of post-COVID-19 MG. In this article, we present a case of post-COVID-19 MG and a concise review of other reported cases. An 83-year-old Caucasian male with a medical history of atrial fibrillation status post-ablation and non-ischemic cardiomyopathy was initially admitted for COVID-19 pneumonia. He was treated with remdesivir, convalescent plasma, and supplemental oxygen therapy but did not require invasive mechanical intubation. One month after discharge, he started experiencing fatigue with muscle weakness and progressive dyspnea. He progressed to develop dysphonia, especially at the end of the day. After extensive workup, he was diagnosed with MG with a positive antibody against the AChR. The chronological events of developing slowly worsening muscular weakness after recovering from COVID-19 infection and positive AChR antibody led to the diagnosis of post-COVID-19 new-onset MG. Post-COVID-19 fatigue, long-term use of steroids, and intensive care unit-related physical deconditioning ....